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Therefore, it truly is conceivable that the JIP1/three → ASK1 → MKK4 signaling module may serve as a conduit to activate JNK when endosomal trafficking is disrupted and glucose stages tumble throughout the early stages of methuosis. Our finding that MKK4 was robustly activated with the methuosis-inducing IPPs, MOMIPP and 2q, and fewer s